A New Role for Eosinophils
EOSINOPHILS: I call them EO’S for short
When the body is having an allergic reaction it produces eosinophils. Due to a 2007 technological breakthrough scientists were, for the first time, able to see that eosinophils regulate pulmonary TH-2 immune responses. Thus an allergic response precedes and indeed initiates the subsequent immune response. By clearing for the allergy we can either avoid or turn off the immune response.
This has remarkable implications. It basically says that any time we see words like IgE, eosinophils, allergen, antigen, antibody, autoantigens, autoantibodies we are dealing with an ailment, illness, or disease that is AN ALLEGY. After 10 years of clinical case studies, I know that allergic reactions can be eliminated. DNA Allergy Relief enables you to treat and eliminate your own allergies.
A 2008 article entitled Allergic Pulmonary Inflammation said: eosinophils elicit a TH-2 immune response in the lungs after allergen challenge. This means that pulmonary (lung) inflammation, and inflammation in general, is an Allergic response, an ALLERGY!
+++ REMINDER: IgE = EEE for ALLERGY! EEE stands for eosinophils.
Now you may understand this research study title:
IgE-Mediates Reactions to
Auto-antigens in Allergic Diseases
“What actually happens, according to Dr. Lee and his team, is that very early in the development of an allergy, the body begins gathering eosinophils in the affected area. When enough are present, they summon T cells to direct the body’s immune response. Dr. Lee’s paper suggests T cells don’t act until beckoned by eosinophils (IgE) the opposite of traditional thinking.”
“A new paradigm has emerged describing eosinophils as initial responders to cell death/tissue damage that are a part of remodeling/repair processes and, more importantly, significant contributors to localized innate and acquired immune responses as well as systemic adaptive immunity.” “Eosinophils not only participate in release of granule proteins, lipid mediators, reactive oxygen species, cytokines, and growth factors but also function through complex cell-cell interactions to elicit chronic T helper 2 (TH-2) inflammation…” (Mayo Clinic Arizona pubmed ref)
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